By Anna R. Hemnes
This booklet discusses general sex-related ameliorations in lung constitution and serve as and the function those adjustments play in lung disorder. New examine at the results of intercourse hormone signaling on particular mobile forms of the lung has all started to bare how those hormones may well force or hinder lung sickness. Expertly written chapters study the consequences of intercourse hormones on general pulmonary constitution and serve as, hormone signaling in lung healthiness, and particular ailments akin to power obstructive pulmonary sickness, bronchial asthma, pulmonary high blood pressure, and lung melanoma. Gender, intercourse Hormones, and respiration disorder: A accomplished Guide specializes in our present knowing and the gaps in examine, with feedback for destiny instructions and implications for treatment. This booklet is an invaluable reference for pulmonologists and researchers and should urged extra inquiry aimed toward bettering total lung overall healthiness.
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Additional info for Gender, Sex Hormones and Respiratory Disease: A Comprehensive Guide
Sample text
DHEA-S can be converted back to the slightly more bioactive DHEA by steroid sulfatase [30]. DHEA then is hydroxylated to androstenedione via 3β-HSD. Of note, androstenedione can also be generated from progesterone. Androstenedione is then oxidized to testosterone by 17β-hydroxysteroid dehydrogenase (17β-HSD). In a next step, testosterone is converted to the more active androgen metabolite, dihydrotestosterone (DHT) by the enzyme 5α-reductase [31]. L. Frump and T. Lahm Fig. 1 Simplified overview of sex hormone steroidogenesis.
Menstrual cycle phase and running economy. Medicine and Science in Sport and Exercise. 1997;29:1609–1618. Chapter 2 Sex Hormone Signaling in the Lung in Health and Disease: Airways, Parenchyma, and Pulmonary Vasculature Andrea L. Frump and Tim Lahm Abstract Gender and sex differences have been implicated in many major lung diseases, such as asthma, chronic obstructive pulmonary disease, cystic fibrosis, pulmonary arterial hypertension, and lung cancer. Sex differences and sex hormonemediated effects emerge in the lung as early as during prenatal development and persist well into adulthood.
94. Babcock MA, Pegelow DF, McClaran SR, Suman OE, Dempsey JA. Contribution of diaphragmatic power output to exercise-induced diaphragm fatigue. J Appl Physiol. 1995; 78:1710–9. 95. Effects of exercise on normoxia and acute hypoxia on respiratory muscle metabolites. J Appl Physiol. 1986;60:1274–83. 96. Effect of N-acetylcysteine on diaphragm fatigue. J Appl Physiol. 1990;68:2107–13. 97. Diaphragmatic fatigue after exercise in healthy human subjects. Am Rev Respir Dis. 1993;148:1571–5. 98. Slower fatigue and faster recovery of the adductor pollicis muscle in women matched for strength with men.