By Ciba Foundation
Content:
Chapter 1 creation (pages 1–3): A. T. Diplock
Chapter 2 nutrition E as an Antioxidant in vitro and in vivo (pages 4–18): G. W. Burton, okay. H. Cheeseman, T. Doba, okay. U. Ingold and T. F. Slater
Chapter three unfastened Radical defense: Why diet E, no longer diet C, ??Carotene or Glutathione? (pages 19–44): R. L. Wilson
Chapter four The position of diet E in organic Membranes (pages 45–55): A. T. Diplock
Chapter five nutrition E, physical exertion and Tissue Oxidative harm (pages 56–69): A. T. Quintanilha and L. Packer
Chapter 6 Tocopherol content material of Adipose Tissue from nutrition E?Deficient people (pages 70–91): Herbert J. Kayden
Chapter 7 Neuropathological reviews of continual diet E Deficiency in Mammals together with people (pages 92–105): James S. Nelson
Chapter eight nutrition E and Neurological functionality: Abetalipoproteinaemia and different problems of fats Absorption (pages 106–129): D. P. R Muller, J. okay. Lloyd and O. H. Wolff
Chapter nine The influence of diet E on Rabbit Neutrophil Activation (pages 130–146): Joan E. Lafuze, Steven J. Weisman, Leah M. Ingraham, Charles J. Butterick, Louise A. Alpert and Robert L. Baehner
Chapter 10 nutrition E and Retrolental Fibroplasia: Prevention of great Ocular Sequelae (pages 165–185): Neil N. Finer, Kathrine L. Peters, Reid F. Schindler and Garry D. Grant
Chapter eleven diet E and Retrolental Fibroplasia: Ultrastructural Mechanism of medical Efficacy (pages 165–185): Helen Hittner, M. Frank and L. Kretzer
Chapter 12 protecting impression of diet E on Intraventricular Haemorrhage within the baby (pages 186–200): Malcolm L. Chiswick, Mary Johnson, Cynthia Woodhall, Maureen Gowland, Jacqueline Davies, Nancy Toner and Douglas Sims
Chapter thirteen Experimental types for dietary Myopathy (pages 201–223): C. H. McMurray, D. A. Rice and S. Kennedy
Chapter 14 diet E and Skeletal Muscle (pages 224–248): M. J. Jackson, D. A. Jones and R. H. T. Edwards
Chapter 15 remaining comments (pages 249–250): A. T. Diplock
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Extra resources for Ciba Foundation Symposium 101 - Biology of Vitamin E
Example text
F. Slater for permission to refer to unpublished results. REFERENCES Allison AC, Eugui 1982 A radical interpretation of immunity to malarial parasites. Lancet 2~1431-1433 Baumann CA, Steenbock H 1933 Fat-soluble vitamins. XXXVII. The stability of carotene solutions. J Biol Chem 101:561-572 Blake DR, Hall ND, Bacon PA, Dieppe PA, Halliwell B, Gutteridge JMC 1981 The importance of iron in rheumatoid disease. Lancet 2:1142-1144 Brugh M 1977 Butylated hydroxytoluene protects chickens exposed to Newcastle disease virus.
Kayden: In my paper I shall show results on different rates of exchange of vitamin E between plasma lipoproteins and erythrocytes. There doesn’t seem to be a specific protein receptor for vitamin E; I don’t know, in fact, what governs the rate of transfer of vitamin E from plasma across into tissue membranes. Edwards: There is a reciprocal, beneficial effect of selenium and vitamin E in deficient animals. The selenium effect is apparently related to glutathione peroxidase, which is a selenoenzyme, but is there any other way in which selenium has effects, perhaps by interacting with the cascade described by Robin Willson?
Under these conditions, intracellular concentrations of 0;- and H202 can be expected to rise, with the danger that oxidant-sensitive intracellular macromolecules may be subject to attack. Furthermore, interaction of 0;- with H20 by a reaction of the type: 0;- + H202 + 0 2 + OH. $. ) that are highly reactive and likely to cause widespread damage. In living cells at neutral pH, the Haber-Weiss reaction will be very slow, but it might be catalysed by metal ions, such as Fe2+, so that significant levels of intracellular OH.