Ciba Foundation Symposium 101 - Biology of Vitamin E by Ciba Foundation

By Ciba Foundation

Chapter 1 creation (pages 1–3): A. T. Diplock
Chapter 2 nutrition E as an Antioxidant in vitro and in vivo (pages 4–18): G. W. Burton, okay. H. Cheeseman, T. Doba, okay. U. Ingold and T. F. Slater
Chapter three unfastened Radical defense: Why diet E, no longer diet C, ??Carotene or Glutathione? (pages 19–44): R. L. Wilson
Chapter four The position of diet E in organic Membranes (pages 45–55): A. T. Diplock
Chapter five nutrition E, physical exertion and Tissue Oxidative harm (pages 56–69): A. T. Quintanilha and L. Packer
Chapter 6 Tocopherol content material of Adipose Tissue from nutrition E?Deficient people (pages 70–91): Herbert J. Kayden
Chapter 7 Neuropathological reviews of continual diet E Deficiency in Mammals together with people (pages 92–105): James S. Nelson
Chapter eight nutrition E and Neurological functionality: Abetalipoproteinaemia and different problems of fats Absorption (pages 106–129): D. P. R Muller, J. okay. Lloyd and O. H. Wolff
Chapter nine The influence of diet E on Rabbit Neutrophil Activation (pages 130–146): Joan E. Lafuze, Steven J. Weisman, Leah M. Ingraham, Charles J. Butterick, Louise A. Alpert and Robert L. Baehner
Chapter 10 nutrition E and Retrolental Fibroplasia: Prevention of great Ocular Sequelae (pages 165–185): Neil N. Finer, Kathrine L. Peters, Reid F. Schindler and Garry D. Grant
Chapter eleven diet E and Retrolental Fibroplasia: Ultrastructural Mechanism of medical Efficacy (pages 165–185): Helen Hittner, M. Frank and L. Kretzer
Chapter 12 protecting impression of diet E on Intraventricular Haemorrhage within the baby (pages 186–200): Malcolm L. Chiswick, Mary Johnson, Cynthia Woodhall, Maureen Gowland, Jacqueline Davies, Nancy Toner and Douglas Sims
Chapter thirteen Experimental types for dietary Myopathy (pages 201–223): C. H. McMurray, D. A. Rice and S. Kennedy
Chapter 14 diet E and Skeletal Muscle (pages 224–248): M. J. Jackson, D. A. Jones and R. H. T. Edwards
Chapter 15 remaining comments (pages 249–250): A. T. Diplock

Show description

Read or Download Ciba Foundation Symposium 101 - Biology of Vitamin E PDF

Similar biology books

Biology and Regulation of Blood-Tissue Barriers

This booklet was once written by way of many awesome investigators who've spent a long time to check varied facets of blood‑tissue barrier functionality. they've got summarized many of the newest and interesting improvement of their fields of analysis together with the blood‑brain barrier, the blood‑retinal barrier, the intestine barrier, the blood‑biliary barrier, the blood‑follicle barrier, the blood‑epididymis barrier, the blood‑testis barrier, the tight junction barrier regularly in addition to obstacles within the woman reproductive tract.

Extra resources for Ciba Foundation Symposium 101 - Biology of Vitamin E

Example text

F. Slater for permission to refer to unpublished results. REFERENCES Allison AC, Eugui 1982 A radical interpretation of immunity to malarial parasites. Lancet 2~1431-1433 Baumann CA, Steenbock H 1933 Fat-soluble vitamins. XXXVII. The stability of carotene solutions. J Biol Chem 101:561-572 Blake DR, Hall ND, Bacon PA, Dieppe PA, Halliwell B, Gutteridge JMC 1981 The importance of iron in rheumatoid disease. Lancet 2:1142-1144 Brugh M 1977 Butylated hydroxytoluene protects chickens exposed to Newcastle disease virus.

Kayden: In my paper I shall show results on different rates of exchange of vitamin E between plasma lipoproteins and erythrocytes. There doesn’t seem to be a specific protein receptor for vitamin E; I don’t know, in fact, what governs the rate of transfer of vitamin E from plasma across into tissue membranes. Edwards: There is a reciprocal, beneficial effect of selenium and vitamin E in deficient animals. The selenium effect is apparently related to glutathione peroxidase, which is a selenoenzyme, but is there any other way in which selenium has effects, perhaps by interacting with the cascade described by Robin Willson?

Under these conditions, intracellular concentrations of 0;- and H202 can be expected to rise, with the danger that oxidant-sensitive intracellular macromolecules may be subject to attack. Furthermore, interaction of 0;- with H20 by a reaction of the type: 0;- + H202 + 0 2 + OH. $. ) that are highly reactive and likely to cause widespread damage. In living cells at neutral pH, the Haber-Weiss reaction will be very slow, but it might be catalysed by metal ions, such as Fe2+, so that significant levels of intracellular OH.

Download PDF sample

Rated 4.01 of 5 – based on 31 votes