By Moore R. L.
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Extra info for An Extension of the Theorem That No Countable Point Set Is Perfect
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P. MacDermott 41. 8 g/day (800 mg tablet) for the treatment of moderately active ulcerative colitis: the ASCEND II trial. Am J Gastroenterol 100(11):2478–2485 42. Marteau P, Probert CS, Lindgren S, Gassul M, Tan TG, Dignass A et al (2005) Combined oral and enema treatment with Pentasa (mesalazine) is superior to oral therapy alone in patients with extensive mild/moderate active ulcerative colitis: a randomised, double blind, placebo controlled study. Gut 54(7):960–965 43. Truelove SC, Watkinson G, Draper G (1962) Comparison of corticosteroid and sulphasalazine therapy in ulcerative colitis.
Thus, to be able to compare reported concordance rates in different twin studies, the proband concordance should be used. Tysk et al. observed proband concordance rates of 58 % and 4 % in monozygotic and dizygotic twins with Crohn’s disease, respectively, reflecting the pronounced genetic predisposition [92]. The corresponding figures for twins with ulcerative colitis were 6 % and 0 %, respectively. Orholm et al. later confirmed these findings in the Danish cohort, where proband concordance rates of 58 % and 0 % were observed in monozygotic and dizygotic twins with Crohn’s disease, respectively [93].
On the whole, the estimated relative risk to a sibling λS (lambda) of a patient with Crohn’s disease is 13–36 and for ulcerative colitis 7–17 [25]. This compares with a λS (lambda) of 9 for schizophrenia, 15 for type 1 diabetes, and 500 for cystic fibrosis [25]. However, from a clinical perspective the relative risk of developing IBD is of less interest. It is the absolute risk of IBD in first-degree relatives and especially in the patient’s offspring that is requested. There are limited studies addressing this clinically relevant question, and quoted absolute risks differ between the studies.